Follow
'However, these tumours often progress without therapy, which suggests the activation of mechanisms that trigger functional impairment of, or resistance to, antitumour immune responses. Indeed, a recent study showed that cyclic GMP–AMP synthase (cGAS)–stimulator of interferon genes (STING) activation in triple-negative breast cancer (TNBC) cells with chromosomal instability induced an interleukin-6 (IL-6)-dependent inflammatory response that promoted the survival of cancer cells'
